Project description
Epidermolysis bullosa (EB) is a severe genetic disease characterised by skin fragility, chronic wounds, inflammation, and scarring, and it can also affect the oral mucosa. In this project, we are comparing healthy and EB-affected oral and skin tissues to understand why scarless healing is lost and which cellular programs drive fibrosis and persistent inflammation.
Our long-term goal is to uncover mechanisms that can support more personalised diagnostics and therapies for chronic wounds and scarring disorders, including EB.